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Wednesday, May 28, 2014

Obesity Homeopathy Specialist Doctor Treatment Clinic at Chennai, Pondicherry, Cuddalore, Villupuram, Panruti, Tamilnadu, India,





   Obesity Obesity in absolute terms is an increase of body fatty tissue mass. In a practical setting it is difficult to measure this directly, and obesity is typically measured by BMI (body mass index) and in terms of its distribution through waist circumference or waist-hip circumference ratio measurements. In addition, the presence of obesity needs to be evaluated in the context of other risk factors and comorbidities (other medical conditions that could influence risk of complications)  BMI Body mass index or BMI is a simple and widely used method for estimating body fat mass. BMI is an accurate reflection of body fat percentage in the majority of the adult population, but is less accurate in situations that affect body composition such as in body builders and pregnancy BMI is calculated by dividing the subject's weight by the square of his/her height, typically expressed either in metric or US "Customary" units: Metric: BMI = kg / m2 Where kg is the subject's weight in kilograms and m is the subject's height in metres. US/Customary and imperial: BMI = lb * 703 / in2 Where lb is the subject's weight in pounds and in is the subject's height in inches The most commonly used definitions, established by the WHO in 1997 and published in 2000, provide the following values:  •	A BMI less than 18.5 is underweight •	A BMI of 18.5–24.9 is normal weight •	A BMI of 25.0–29.9 is overweight •	A BMI of 30.0–34.9 is class I obesity •	A BMI of 35.0-39.9 is class II obesity •	A BMI of > 40.0 is class III obesity or severe / morbidly obese •	A BMI of 35.0 or higher in the presence of at least one other significant comorbidity is also classified by some bodies as morbid obesity.  Causes Most researchers agree that a combination of excessive calorie consumption and a sedentary lifestyle are the primary causes of obesity in the majority of the population. Other less well established or minor influences include genetic causes, medical and psychiatric illnesses, and microbiological causes.  A review identifies ten other possibly under investigated causes for recently increasing rates of obesity:  (1) Insufficient sleep  (2) Endocrine disruptors - food substances that interfere with lipid metabolism,  (3) decreased variability in ambient temperature,  (4) decreased rates of smoking, which suppresses appetite,  (5) increased use of medication that leads to weight gain,  (6) Increased distribution of ethnic and age groups that tend to be heavier,  (7) Pregnancy at a later age,  (8) Intrauterine and intergenerational effects,  (9) Positive natural selection of people with a higher BMI,  (10) Assortative mating, heavier people tending to form relationships with each other.   Dietary 	Despite the widespread availability of nutritional information in schools, doctors' offices, on the internet and on product packaging, it is evident that overeating remains a substantial problem.   Sedentary lifestyle 	An increasingly sedentary lifestyle plays a significant role in obesity. There has been a trend toward decreased physical activity due in part to increasingly mechanized forms of work, changing modes of transportation, and increasing urbanization. Studies in children and adults have found an association between the number of hours of television watched and the prevalence of obesity. Driving one's children to school also decreases the amount of exercise that these children get and has led to calls for reduced car use around schools. An association between leisure time activity and obesity has been found.   Genetics 	Like many other medical conditions, obesity is the result of interplay between genetic and environmental factors. Polymorphisms in various genes controlling appetite, metabolism, and adipokine release may predispose to obesity when sufficient calories are present. Obesity is a major feature in a number of rare genetic conditions: Prader-Willi syndrome, Bardet-Biedl syndrome, MOMO syndrome, leptin receptor mutations, and melanocortin receptor mutations. In a people with early-onset severe obesity (defined by an onset before ten years of age and body mass index over three standard deviations above normal), 7% harbour a single locus mutation. Apart from the above syndromes, an association has been found between an FTO gene polymorphism and weight. The 16% of adults in the study who were homozygous for this allele weighed about 3 kilograms more then those who had not inherited this trait and subsequently had a 1.6 fold greater rate of obesity.  	On a population level, the thrifty gene hypothesis postulates that certain ethnic groups may be more prone to obesity when exposed to an equivalent environment. Their ability to take advantage of rare periods of abundance by storing energy as fat would be advantageous during times of varying food availability. Individuals with greater adipose reserves would be more likely survive famine. This tendency to store fat however would be maladaptive in societies with stable food supplies.   Medical illness 	Certain physical and mental illnesses and the pharmaceutical substances used to treat them can increase one's risk of obesity. Medical illnesses that increase obesity risk include several rare genetic syndromes (listed above) as well as some congenital or acquired conditions: (1) hypothyroidism, (2) Cushing's syndrome, (3) growth hormone deficiency, and (4) eating disorders such bulimia nervosa, binge eating disorder and compulsive overeating. 	Certain medications may cause weight gain and or negative changes in body composition, such as steroids, atypical antipsychotics, some fertility medication, insulin and sulfonylureas.  Microbiological 	The role of bacteria colonizing the digestive tract in the development of obesity has recently become the subject of investigation. Bacteria participate in digestion (especially of fatty acids and polysaccharides), and alterations in the proportion of particular strains of bacteria may explain why certain people are more prone to weight gain than others. Human digestive tract bacteria are generally either member of the phyla of bacteroidetes or of firmicutes. In obese people, there is a relative abundance of firmicutes (which cause relatively high energy absorption), which is restored by weight loss. From these results it cannot be concluded whether this imbalance is the cause or effect of obesity.   Social determinants 	The correlation between social class and BMI is inconsistent. Comparing net worth with BMI found obese Americanians approximately half as wealthy as thin ones. When income differentials were factored out, the inequity persisted. A tendency to rely on fast food is seen as one of the reasons why this result occurred. Another study found women who married into a higher social class are thinner than women who married into a lower social class. A Health Survey however found the exact opposite. It found that men from lower middle income households were less likely to be obese than were those in the highest income households and women from middle income households had the highest rates of obesity.   Obesity  Homeopathy Treatment Symptomatic Homeopathy works well for Obesity,  It helps to prevent further recurrence also. So its good to consult a experienced Homeopathy physician without any hesitation. Homoeopathic system of medicine are very effective for obesity,  regular intake of medicine is very much helpful to control and reduce the weight without any side effect   Whom to contact for Obesity Treatment  Dr.Senthil Kumar Treats many cases of Obesity, In his medical professional experience with successful results. Many patients get relief after taking treatment from Dr.Senthil Kumar.  Dr.Senthil Kumar visits Chennai at Vivekanantha Homeopathy Clinic, Velachery, Chennai 42. To get appointment please call 9786901830, +91 94430 54168 or mail to consult.ur.dr@gmail.com,    For more details & Consultation Feel free to contact us. Vivekanantha Clinic Consultation Champers at Chennai:- 9786901830  Panruti:- 9443054168  Pondicherry:- 9865212055 (Camp) Mail : consult.ur.dr@gmail.com, homoeokumar@gmail.com   For appointment please Call us or Mail Us  For appointment: SMS your Name -Age – Mobile Number - Problem in Single word - date and day - Place of appointment (Eg: Rajini – 30 - 99xxxxxxx0 – Obesity – 21st Oct, Sunday - Chennai ), You will receive Appointment details through SMS





Obesity
Obesity in absolute terms is an increase of body fatty tissue mass. In a practical setting it is difficult to measure this directly, and obesity is typically measured by BMI (body mass index) and in terms of its distribution through waist circumference or waist-hip circumference ratio measurements. In addition, the presence of obesity needs to be evaluated in the context of other risk factors and comorbidities (other medical conditions that could influence risk of complications)

BMI
Body mass index or BMI is a simple and widely used method for estimating body fat mass. BMI is an accurate reflection of body fat percentage in the majority of the adult population, but is less accurate in situations that affect body composition such as in body builders and pregnancy
BMI is calculated by dividing the subject's weight by the square of his/her height, typically expressed either in metric or US "Customary" units:
Metric: BMI = kg / m2
Where kg is the subject's weight in kilograms and m is the subject's height in metres.
US/Customary and imperial: BMI = lb * 703 / in2
Where lb is the subject's weight in pounds and in is the subject's height in inches
The most commonly used definitions, established by the WHO in 1997 and published in 2000, provide the following values:
  • A BMI less than 18.5 is underweight
  • A BMI of 18.5–24.9 is normal weight
  • A BMI of 25.0–29.9 is overweight
  • A BMI of 30.0–34.9 is class I obesity
  • A BMI of 35.0-39.9 is class II obesity
  • A BMI of > 40.0 is class III obesity or severe / morbidly obese
  • A BMI of 35.0 or higher in the presence of at least one other significant comorbidity is also classified by some bodies as morbid obesity.

Causes
Most researchers agree that a combination of excessive calorie consumption and a sedentary lifestyle are the primary causes of obesity in the majority of the population. Other less well established or minor influences include genetic causes, medical and psychiatric illnesses, and microbiological causes.
A review identifies ten other possibly under investigated causes for recently increasing rates of obesity:
(1) Insufficient sleep
(2) Endocrine disruptors - food substances that interfere with lipid metabolism,
(3) decreased variability in ambient temperature,
(4) decreased rates of smoking, which suppresses appetite,
(5) increased use of medication that leads to weight gain,
(6) Increased distribution of ethnic and age groups that tend to be heavier,
(7) Pregnancy at a later age,
(8) Intrauterine and intergenerational effects,
(9) Positive natural selection of people with a higher BMI,
(10) Assortative mating, heavier people tending to form relationships with each other.

Dietary
v  Despite the widespread availability of nutritional information in schools, doctors' offices, on the internet and on product packaging, it is evident that overeating remains a substantial problem.

Sedentary lifestyle
v  An increasingly sedentary lifestyle plays a significant role in obesity. There has been a trend toward decreased physical activity due in part to increasingly mechanized forms of work, changing modes of transportation, and increasing urbanization. Studies in children and adults have found an association between the number of hours of television watched and the prevalence of obesity. Driving one's children to school also decreases the amount of exercise that these children get and has led to calls for reduced car use around schools. An association between leisure time activity and obesity has been found.

Genetics
v  Like many other medical conditions, obesity is the result of interplay between genetic and environmental factors. Polymorphisms in various genes controlling appetite, metabolism, and adipokine release may predispose to obesity when sufficient calories are present. Obesity is a major feature in a number of rare genetic conditions: Prader-Willi syndrome, Bardet-Biedl syndrome, MOMO syndrome, leptin receptor mutations, and melanocortin receptor mutations. In a people with early-onset severe obesity (defined by an onset before ten years of age and body mass index over three standard deviations above normal), 7% harbour a single locus mutation. Apart from the above syndromes, an association has been found between an FTO gene polymorphism and weight. The 16% of adults in the study who were homozygous for this allele weighed about 3 kilograms more then those who had not inherited this trait and subsequently had a 1.6 fold greater rate of obesity.
v  On a population level, the thrifty gene hypothesis postulates that certain ethnic groups may be more prone to obesity when exposed to an equivalent environment. Their ability to take advantage of rare periods of abundance by storing energy as fat would be advantageous during times of varying food availability. Individuals with greater adipose reserves would be more likely survive famine. This tendency to store fat however would be maladaptive in societies with stable food supplies.

Medical illness
v  Certain physical and mental illnesses and the pharmaceutical substances used to treat them can increase one's risk of obesity. Medical illnesses that increase obesity risk include several rare genetic syndromes (listed above) as well as some congenital or acquired conditions: (1) hypothyroidism, (2) Cushing's syndrome, (3) growth hormone deficiency, and (4) eating disorders such bulimia nervosa, binge eating disorder and compulsive overeating.
v  Certain medications may cause weight gain and or negative changes in body composition, such as steroids, atypical antipsychotics, some fertility medication, insulin and sulfonylureas.

Microbiological
v  The role of bacteria colonizing the digestive tract in the development of obesity has recently become the subject of investigation. Bacteria participate in digestion (especially of fatty acids and polysaccharides), and alterations in the proportion of particular strains of bacteria may explain why certain people are more prone to weight gain than others. Human digestive tract bacteria are generally either member of the phyla of bacteroidetes or of firmicutes. In obese people, there is a relative abundance of firmicutes (which cause relatively high energy absorption), which is restored by weight loss. From these results it cannot be concluded whether this imbalance is the cause or effect of obesity.

Social determinants
v  The correlation between social class and BMI is inconsistent. Comparing net worth with BMI found obese Americanians approximately half as wealthy as thin ones. When income differentials were factored out, the inequity persisted. A tendency to rely on fast food is seen as one of the reasons why this result occurred. Another study found women who married into a higher social class are thinner than women who married into a lower social class. A Health Survey however found the exact opposite. It found that men from lower middle income households were less likely to be obese than were those in the highest income households and women from middle income households had the highest rates of obesity.

Obesity  Homeopathy Treatment
Symptomatic Homeopathy works well for Obesity,  It helps to prevent further recurrence also. So its good to consult a experienced Homeopathy physician without any hesitation. Homoeopathic system of medicine are very effective for obesity,  regular intake of medicine is very much helpful to control and reduce the weight without any side effect


Whom to contact for Obesity Treatment
Dr.Senthil Kumar Treats many cases of Obesity, In his medical professional experience with successful results. Many patients get relief after taking treatment from Dr.Senthil Kumar.  Dr.Senthil Kumar visits Chennai at Vivekanantha Homeopathy Clinic, Velachery, Chennai 42. To get appointment please call 9786901830, +91 94430 54168 or mail to consult.ur.dr@gmail.com,


For more details & Consultation Feel free to contact us.
   Obesity Obesity in absolute terms is an increase of body fatty tissue mass. In a practical setting it is difficult to measure this directly, and obesity is typically measured by BMI (body mass index) and in terms of its distribution through waist circumference or waist-hip circumference ratio measurements. In addition, the presence of obesity needs to be evaluated in the context of other risk factors and comorbidities (other medical conditions that could influence risk of complications)  BMI Body mass index or BMI is a simple and widely used method for estimating body fat mass. BMI is an accurate reflection of body fat percentage in the majority of the adult population, but is less accurate in situations that affect body composition such as in body builders and pregnancy BMI is calculated by dividing the subject's weight by the square of his/her height, typically expressed either in metric or US "Customary" units: Metric: BMI = kg / m2 Where kg is the subject's weight in kilograms and m is the subject's height in metres. US/Customary and imperial: BMI = lb * 703 / in2 Where lb is the subject's weight in pounds and in is the subject's height in inches The most commonly used definitions, established by the WHO in 1997 and published in 2000, provide the following values:  •	A BMI less than 18.5 is underweight •	A BMI of 18.5–24.9 is normal weight •	A BMI of 25.0–29.9 is overweight •	A BMI of 30.0–34.9 is class I obesity •	A BMI of 35.0-39.9 is class II obesity •	A BMI of > 40.0 is class III obesity or severe / morbidly obese •	A BMI of 35.0 or higher in the presence of at least one other significant comorbidity is also classified by some bodies as morbid obesity.  Causes Most researchers agree that a combination of excessive calorie consumption and a sedentary lifestyle are the primary causes of obesity in the majority of the population. Other less well established or minor influences include genetic causes, medical and psychiatric illnesses, and microbiological causes.  A review identifies ten other possibly under investigated causes for recently increasing rates of obesity:  (1) Insufficient sleep  (2) Endocrine disruptors - food substances that interfere with lipid metabolism,  (3) decreased variability in ambient temperature,  (4) decreased rates of smoking, which suppresses appetite,  (5) increased use of medication that leads to weight gain,  (6) Increased distribution of ethnic and age groups that tend to be heavier,  (7) Pregnancy at a later age,  (8) Intrauterine and intergenerational effects,  (9) Positive natural selection of people with a higher BMI,  (10) Assortative mating, heavier people tending to form relationships with each other.   Dietary 	Despite the widespread availability of nutritional information in schools, doctors' offices, on the internet and on product packaging, it is evident that overeating remains a substantial problem.   Sedentary lifestyle 	An increasingly sedentary lifestyle plays a significant role in obesity. There has been a trend toward decreased physical activity due in part to increasingly mechanized forms of work, changing modes of transportation, and increasing urbanization. Studies in children and adults have found an association between the number of hours of television watched and the prevalence of obesity. Driving one's children to school also decreases the amount of exercise that these children get and has led to calls for reduced car use around schools. An association between leisure time activity and obesity has been found.   Genetics 	Like many other medical conditions, obesity is the result of interplay between genetic and environmental factors. Polymorphisms in various genes controlling appetite, metabolism, and adipokine release may predispose to obesity when sufficient calories are present. Obesity is a major feature in a number of rare genetic conditions: Prader-Willi syndrome, Bardet-Biedl syndrome, MOMO syndrome, leptin receptor mutations, and melanocortin receptor mutations. In a people with early-onset severe obesity (defined by an onset before ten years of age and body mass index over three standard deviations above normal), 7% harbour a single locus mutation. Apart from the above syndromes, an association has been found between an FTO gene polymorphism and weight. The 16% of adults in the study who were homozygous for this allele weighed about 3 kilograms more then those who had not inherited this trait and subsequently had a 1.6 fold greater rate of obesity.  	On a population level, the thrifty gene hypothesis postulates that certain ethnic groups may be more prone to obesity when exposed to an equivalent environment. Their ability to take advantage of rare periods of abundance by storing energy as fat would be advantageous during times of varying food availability. Individuals with greater adipose reserves would be more likely survive famine. This tendency to store fat however would be maladaptive in societies with stable food supplies.   Medical illness 	Certain physical and mental illnesses and the pharmaceutical substances used to treat them can increase one's risk of obesity. Medical illnesses that increase obesity risk include several rare genetic syndromes (listed above) as well as some congenital or acquired conditions: (1) hypothyroidism, (2) Cushing's syndrome, (3) growth hormone deficiency, and (4) eating disorders such bulimia nervosa, binge eating disorder and compulsive overeating. 	Certain medications may cause weight gain and or negative changes in body composition, such as steroids, atypical antipsychotics, some fertility medication, insulin and sulfonylureas.  Microbiological 	The role of bacteria colonizing the digestive tract in the development of obesity has recently become the subject of investigation. Bacteria participate in digestion (especially of fatty acids and polysaccharides), and alterations in the proportion of particular strains of bacteria may explain why certain people are more prone to weight gain than others. Human digestive tract bacteria are generally either member of the phyla of bacteroidetes or of firmicutes. In obese people, there is a relative abundance of firmicutes (which cause relatively high energy absorption), which is restored by weight loss. From these results it cannot be concluded whether this imbalance is the cause or effect of obesity.   Social determinants 	The correlation between social class and BMI is inconsistent. Comparing net worth with BMI found obese Americanians approximately half as wealthy as thin ones. When income differentials were factored out, the inequity persisted. A tendency to rely on fast food is seen as one of the reasons why this result occurred. Another study found women who married into a higher social class are thinner than women who married into a lower social class. A Health Survey however found the exact opposite. It found that men from lower middle income households were less likely to be obese than were those in the highest income households and women from middle income households had the highest rates of obesity.   Obesity  Homeopathy Treatment Symptomatic Homeopathy works well for Obesity,  It helps to prevent further recurrence also. So its good to consult a experienced Homeopathy physician without any hesitation. Homoeopathic system of medicine are very effective for obesity,  regular intake of medicine is very much helpful to control and reduce the weight without any side effect   Whom to contact for Obesity Treatment  Dr.Senthil Kumar Treats many cases of Obesity, In his medical professional experience with successful results. Many patients get relief after taking treatment from Dr.Senthil Kumar.  Dr.Senthil Kumar visits Chennai at Vivekanantha Homeopathy Clinic, Velachery, Chennai 42. To get appointment please call 9786901830, +91 94430 54168 or mail to consult.ur.dr@gmail.com,    For more details & Consultation Feel free to contact us. Vivekanantha Clinic Consultation Champers at Chennai:- 9786901830  Panruti:- 9443054168  Pondicherry:- 9865212055 (Camp) Mail : consult.ur.dr@gmail.com, homoeokumar@gmail.com   For appointment please Call us or Mail Us  For appointment: SMS your Name -Age – Mobile Number - Problem in Single word - date and day - Place of appointment (Eg: Rajini – 30 - 99xxxxxxx0 – Obesity – 21st Oct, Sunday - Chennai ), You will receive Appointment details through SMSVivekanantha Clinic Consultation Champers at
Chennai:- 9786901830
Panruti:- 9443054168
Pondicherry:- 9865212055 (Camp)

For appointment please Call us or Mail Us

For appointment: SMS your Name -Age – Mobile Number - Problem in Single word - date and day - Place of appointment (Eg: Rajini – 30 - 99xxxxxxx0 – Obesity – 21st Oct, Sunday - Chennai ), You will receive Appointment details through SMS







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Clinic & Camp Clinics

Vivekanantha Homoeo Clinic & Psychological Counselling Center

Dr.Senthil Kumar’s Consultation Schedule

Chennai

Head Office

Monday to Saturday:- 10.00am to 12.30pm &

5.00pm to 8.30pm

Sunday: - 10.30am to 12.30pm

(Consultation by Appointment only)

For Appointment

Please call: 09443054168,

Paramount Park

(Dr Plaza) - B Block,

B-12, Second Floor,

Velachery Main Road,

Direct Opposite to Saravana Stores,

Supreme Mobiles upstairs,

Near Vijaya nagar Bus Stand,

Velachery, Chennai 42,

Panruti

Branch Office

Monday(First & Third Monday of Every Month)

10.00am to 12.30pm &

05.30pm to 8.30pm

(Consultation by Appointment only)

For Appointment

Please call: 09443054168,

11, Kuchipalayam Street

(Opposite lane to Boys Hr Sec School), Panruti-607106,

Cuddalore District,

Tamil Nadu, India



Pondicherry

Branch Office

Every Saturday:

11.00am to 02.00pm

(Consultation by Appointment only)

Appointment

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NB:-

Ø We are taking only minimum number of patients per day.

Ø We are allotting 40 to 5o minutes for new patients & 15 to 20 minutes for follow-ups.

Ø So be there at time to avoid unwanted waiting

Ø we concentrate more to patient’s privacy, so we are allotting 40 to 50 minutes/client – “so be there at time”

Ø We treat Many Diseases, so no one can know for what problem you are taking the treatment – So feel free to talk with Doctor and visit the Clinic.

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Please call: 09443054168, 09786901830

Please call the Doctor and explain your problems in short, then SMS your Name – Mobile Number - Problem in Single word - date and day - Place of appointment (Eg: Rajini - 99xxxxxxx0 – Psoriasis – 21st Oct Sunday - Chennai )

You will receive Appointment details through SMS

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Disclaimer: These articles is for information only and should not be used for the diagnosis or treatment of medical conditions. we used all reasonable care in compiling the information but make no warranty as to its accuracy. Consult a doctor or other health care professional for diagnosis and treatment of medical conditions.